Thread: 1 mg Methyltren 3 days a week pretraining safe duration?

Hi

I have 2 vials of mtren left over
I ran an entire vial 2 weeks straight and it was simply too harsh really for me to use to peak strength with, mainly because it fucked my sleep horribly and made me fatigued. Like I would have horrendous 4 hrs broken light sleep, anything waking me up... not restful at all!

Safety wise, how long could I use 1 mg injected mtren Mon Wed Fri?

I read up some data of oral mtren, 100 mcg ED after 2 weeks totally trashed liver functon

If you inject 1 mg ED and the drug has a half life of 6 hrs (iirc) then you will have 125 mg 18 hrs after first injection 62.5 24 hrs after where you would inject another 1000 mcg to get 1062.5 mcg... then peak again 1066 mg.. after the 2 days off you'd have about 4 mcg in you where on Monday you'd pin another 1000 and the cycle repeats

Basically this seems more stressful than 100 mcg ED... FAR more...
Then again, by injecting you avoid the first pass metabolism entirely then you have 2nd metabolism to deal with... also about 70% less compound through liver than via oral ingestion so you have 70% less and avoid the 1st pass... I have seen stories of people doing "3 mg" Mon Wed Fri for 3 wks and getting ALT of 80 AST of 60, nothing bad! I feel this was underdosed stuff though considering how badly 100 mcg wrecks you.

Thoughts?

Injecting doesn't make it less hepatoxic.... I don't get where people get this idea from... It's faster onset and more accurate dosing. That's it. Hormone still passes through liver and causes damage, doesn't matter if it's first, second, etc. The hormone itself damages the liver NOT the order in which it passes through. In theory injecting should be MORE harmful as more hormone actually makes it into the blood vs breakdown via stomach, food in digestion, sensitivity to the hormone causing gastric emptying, etc. Also the added solvent to put a methylated compound in solution damages the liver as well...

Are you sure about this? I have read articles comparing Winstrol Depot to oral Winstrol and there hepatic markers less severely awry in the depot group

Hi

Sorry for late reply

When you take it orally, it goes through a tube from the stomach to the liver where 1st pass metabolism happens
This is avoided when taken I.M
So you have an entire bunch of metabolic pathways avoided
Also not only are you avoiding an entire metabolism of the drug by the liver, 70% less of the compound reaches the liver as if it were taken orally iirc

So you have 70% less compound hitting it and an entire metabolism of the drug removed

But the liver is still what removes the compound from the body. Damage would still occur. Do a test of it! Lol run some IM for a few weeks and blood test. Then wait a while for liver to do recover and do oral for a few weeks and compare results. If IM made the safety margin that much different we wouldn't have orals imo.

Originally Posted by Serotonin101

But the liver is still what removes the compound from the body. Damage would still occur. Do a test of it! Lol run some IM for a few weeks and blood test. Then wait a while for liver to do recover and do oral for a few weeks and compare results. If IM made the safety margin that much different we wouldn't have orals imo.

Orals are more potent mg for mg I thought, precisely because the liver processes more harshly. Also orals clear the body much faster than IM which is important for drug tested sports

Originally Posted by SMonMTS

Orals are more potent mg for mg I thought, precisely because the liver processes more harshly. Also orals clear the body much faster than IM which is important for drug tested sports

I should have specified as you seem confused by my wording. If IM administration of methylated compounds nullified the liver toxicity by that great of a margin then there wouldn't be an oral methylated steroid market as everyone would be injecting their methylated compounds as then they could do it for a longer duration. The difference would most certainly be negligible in my opinion which is why people still orally take methylated compounds vs injecting them.
The methyltrienelone is more than likely dissolved in oil for injection because it makes dosing things on a microgram scale easier as a solution is homogeneous, same distribution of hormone throughout where as making a powder capsule or pressed pill could yield "hot spots" where the concentration is higher. That could end disastrously for something active and potent in the microgram dosing range.

Well regardless of the safety/potency which neither of us really know what is true ;D orals are still gonna be around because you can't dodge drug tests on methtlated compounds injected IM

Basically orals are taken orally because that is what their format was when they were introduced as legitimate white market medical drugs! What average person with anemia etc would want to be pinning anadrol suspension every day when they could take 50 mg ED safely orally for weeks and weeks?

Also injected c-17s make you test positive for aaallooot longer than orals

Also when taken orally it binds to shbg which theoretically can make your injectables more effective but the oral steroid elss effective, injecting it strangely makes c-17s bind much less to SHBG

Here is an interesting argument: https://thinksteroids.com/articles/w...vs-injectable/